Deletion of S. Typhi tolc affects bacterial growth, efflux, adhesion

Antibiotics, Free Full-Text

Frontiers Deletion of Salmonella enterica serovar Typhi tolC reduces bacterial adhesion and invasion toward host cells

AcrAB efflux pump impacts on the survival of adherent-invasive Escherichia coli strain LF82 inside macrophages

The Multidrug Efflux System AcrABZ-TolC Is Essential for Infection of Salmonella Typhimurium by the Flagellum-Dependent Bacteriophage Chi

Frontiers MacAB-TolC Contributes to the Development of Acinetobacter baumannii Biofilm at the Solid–Liquid Interface

Drug resistance and physiological roles of RND multidrug efflux pumps in Salmonella enterica, Escherichia coli and Pseudomonas aeruginosa

Fast bacterial growth reduces antibiotic accumulation and efficacy

AcrAB efflux pump impacts on the survival of adherent-invasive Escherichia coli strain LF82 inside macrophages

Pathogens, Free Full-Text

TolC, but not AcrB, is involved in the invasiveness of multidrug-resistant Salmonella enterica serovar Typhimurium by increasing type III secretion system-1 expression.

A macrophage-based screen identifies antibacterial compounds selective for intracellular Salmonella Typhimurium